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Anacetrapib

Anacetrapib dose-dependently shrinks atherosclerotic lesions by up to 92% and adds to the benefit of atorvastatin (Eur Heart J 2015)

Original title: Anacetrapib reduces progression of atherosclerosis, mainly by reducing non-HDL-cholesterol, improves lesion stability and adds to the beneficial effects of atorvastatin

Eur Heart J · · 7

Kühnast S, van der Tuin SJ, van der Hoorn JW, van Klinken JB, Simic B, Pieterman E, Havekes LM, Landmesser U, Lüscher TF, Willems van Dijk K, Rensen PC, Jukema JW et al.

APOE*3Leiden.CETP mice were fed a diet with ascending anacetrapib doses (0.03, 0.3, 3, or 30 mg/kg/day), atorvastatin 2.4 mg/kg/day alone, or atorvastatin combined with anacetrapib 0.3 mg/kg/day, for 21 weeks. Anacetrapib dose-dependently reduced CETP activity (-59 to -100%, P less than 0.001), decreasing non-HDL-C (-24 to -45%) and increasing HDL-C (+30 to +86%, both P less than 0.001). Atherosclerotic lesion area fell dose-dependently by -41 to -92% (P less than 0.01), lesion severity decreased, and plaque stability index rose; combining anacetrapib with atorvastatin further reduced lesion size by -95% (P less than 0.001). Analysis of covariance showed both anacetrapib itself (P less than 0.05) and non-HDL-C (P less than 0.001), but not HDL-C (P equals 0.76), independently predicted lesion size, indicating the anti-atherogenic effect of anacetrapib and its additive benefit with atorvastatin are driven mainly by lowering non-HDL-C rather than raising HDL-C.

Read the paper (DOI)PubMed

Original abstract

Background: The residual risk that remains after statin treatment supports the addition of other LDL-C-lowering agents and has stimulated the search for secondary treatment targets. Epidemiological studies propose HDL-C as a possible candidate. Cholesteryl ester transfer protein (CETP) transfers cholesteryl esters from atheroprotective HDL to atherogenic (V)LDL. The CETP inhibitor anacetrapib decreases (V)LDL-C by ∼15-40% and increases HDL-C by ∼40-140% in clinical trials. We evaluated the effects of a broad dose range of anacetrapib on atherosclerosis and HDL function, and examined possible additive/synergistic effects of anacetrapib on top of atorvastatin in APOE*3Leiden.CETP mice.

Methods And Results: Mice were fed a diet without or with ascending dosages of anacetrapib (0.03; 0.3; 3; 30 mg/kg/day), atorvastatin (2.4 mg/kg/day) alone or in combination with anacetrapib (0.3 mg/kg/day) for 21 weeks. Anacetrapib dose-dependently reduced CETP activity (-59 to -100%, P < 0.001), thereby decreasing non-HDL-C (-24 to -45%, P < 0.001) and increasing HDL-C (+30 to +86%, P < 0.001). Anacetrapib dose-dependently reduced the atherosclerotic lesion area (-41 to -92%, P < 0.01) and severity, increased plaque stability index and added to the effects of atorvastatin by further decreasing lesion size (-95%, P < 0.001) and severity. Analysis of covariance showed that both anacetrapib (P < 0.05) and non-HDL-C (P < 0.001), but not HDL-C (P = 0.76), independently determined lesion size.

Conclusion: Anacetrapib dose-dependently reduces atherosclerosis, and adds to the anti-atherogenic effects of atorvastatin, which is mainly ascribed to a reduction in non-HDL-C. In addition, anacetrapib improves lesion stability.

anacetrapibmechanisms

Summary written by cetpinhibition.org from the published abstract; figures as published. Page updated 18 August 2026. Methods.