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CETP gene mutations shape LDL and HDL cholesterol from birth through the first year of life (Clin Chim Acta 2009)

Original title: Lipoprotein profiles in children with two common cholesteryl ester transfer protein gene mutations, D442G and I14A, during the first year of life

Clin Chim Acta · · 7

Nagasaka H, Yorifuji T, Momoi T, Yorifuji J, Hirano K, Ota A, Takatani T, Tsukahara H, Takayanagi M, Kobayashi K, Chiba H, Sato Y et al.

In infants with CETP D442G or I14A gene mutations (19 heterozygotes, one D442G/I14A compound heterozygote), 13 non-affected siblings, and 30 healthy controls followed at birth, 3-4 months, and 12 months, CETP mass was 32-70% lower in heterozygotes than controls throughout the year. LDL-cholesterol was 30%, 20%, and 15% lower in heterozygotes at birth, 3-4 months, and 12 months respectively, while HDL-cholesterol, similar to controls at birth, became 10% higher in heterozygotes at 3-4 and 12 months, and ApoE-rich HDL-cholesterol became 50% higher. These effects were most pronounced in the compound heterozygote and absent in non-affected siblings, and CETP mass correlated positively with LDL-C but negatively with HDL-C, showing CETP already determines LDL-C and HDL-C levels within the first year of life.

Read the paper (DOI)PubMed

Original abstract

Background: Hyperalphalipoproteinemia is associated with cholesteryl ester transfer protein (CETP) deficiency in adults but has unclear associations in children.

Methods: We measured lipoproteins in 19 heterozygotes (D442G, n=17; I14A, n=2), one D442G/I14A compound heterozygote, 13 non-affected siblings, and 30 healthy controls at birth, 3-4 months, and 12 months.

Results: CETP mass was 32-70% lower in heterozygotes than in controls throughout the year. Low-density lipoprotein-cholesterol (LDL-C) was lower in heterozygotes than in controls by 30, 20, and 15% at birth, 3-4 months, and 12 months, respectively. High-density lipoprotein-cholesterol (HDL-C) was similar among the groups at birth, but was 10% higher in heterozygotes compared with controls at 3-4 and 12 months. ApoE-rich HDL-C was similar between the two groups at birth, but was 50% higher in heterozygotes than in controls at 3-4 and 12 months. These lipoprotein profile characteristics were prominent in the compound heterozygote but were not found in non-affected siblings. In heterozygotes, CETP mass correlated positively with LDL-C but negatively with HDL-C at 3-4 and 12 months.

Conclusion: CETP is a determinant for LDL-C and HDL-C in CETP-deficient individuals in the first year of life.

childrengeneticsHDL biology

Summary written by cetpinhibition.org from the published abstract; figures as published. Page updated 19 August 2026. Methods.